Reviewers you may be interested in

Re Name

Total: 413

Country: United States

Keywords: cancer, drug

Vladimir Achikyan

Total: 96

Country: Russian Federation

Keywords: Immunology, Rheumatology, inflammatory bowel disease, colitis, musculoskeletal disorders, pain, sleep disorders, HCV, liver disease, HIV, AIDS, Oncology, breast cancer, lung cancer, melanoma, Oncourology, clinical trials, clinical studies, real world data, real world evidence

Anish Singh

Total: 93

Country: India

Keywords: Neuropharmacology, Nephropharmacology, Alzheimer's, Parkinson's disease, Epilepsy, Neuron, Nephron, Cardiovascular, Drug Science

Amir Miri

Total: 90

Country: Iran (the Islamic Republic of)

Keywords: Antibiotics, antibiotic resistance, nanoparticles, nanomedicine, active targeting, passive targeting, surface functionalization, controlled release, microparticles, probiotics, gut microbiota, gastric microbiota, infectious diseases, microbiome, Helicobacter pylori, drug delivery systems, Alternatives to antibiotics.

Xiaohe Jin

Total: 76

Country: 

Keywords: Biosensors; Photosynthesis; Synthetic Biology; Nano(bio)materials; Self-assembly of biomolecular materials; Gene/Drug delivery systems; DNA Nanostructure Scaffolds; Hydrogels; Nanobiotechnology; Cell Imaging.

xinna li

Total: 72

Country: United States

Keywords: I have strong research experience in molecular biology and biochemistry. In 2012, I joined the group of Richard Miller to study the mechanism of aging using longevity mouse models. My work has provided encouraging evidence that ARIs (Aging Rate Indicators) may be altered by genes, CR diet, and drugs in many tissues of many varieties of slow aging mice. The slow aging is attributed to a multitude of causes including mutations (Ames, Snell, GHR-KO, or PAPPA-KO) and CR diet, mice treated with rapamycin (Rapa), acarbose (Aca), 17aE2, and canagliflozin (Cana).These genes and interventions lead to many physiological changes such as 1) an increase in uncoupling protein UCP1 in brown and white adipose tissue (WAT); 2) a change in fat-associated macrophage subsets that leads to diminished production of inflammatory cytokines; 3) an increase in muscle fibronectin type III domain containing 5 (FNDC5) and its cleavage product, the myokine irisin, thought to cause changes in fat cell differentiation; 4) elevated production of hepatic GPLD1 and plasma GPLD1; 5) elevation of hippocampal BDNF and DCX, used as indices of neurogenesis.